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Its relevance to neuropathy centers on three mechanisms: cell migration promotion, neuroinflammation reduction, and direct effects on nervous system repair cells

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Given that CRAT has been recognized as a crucial regulator in the balance of mitochondrial acetyl-CoA, which is the donor of acetyl groups for protein acetylation, the effects of CRAT on the levels of acetyl-CoA and PGC-1α acetylation were thus determined